Leading up to the clinical description in this work, Akinetopsia was described in works as early as 1911, in a description by researchers Poltzl and Redlich; however, there was no research on the location of the lesion or the extent of the symptoms (Zeki, 1991).
case reports describing patient's symptoms in detail
cASE REPORT #1
In 1983, Zihl and his colleagues published a case study which described a patient with defective motion vision. The patient suffered from a bilateral cerebral vascular lesion in the cortex outside the striate area. This case study marked the first clinically recorded case of cerebral motion blindness, or Akinetopsia (Zihl, 1983). The patient had instact visual acuity and visual fields, but was rendered unable to see motion due the bilateral occipito-temporal ischemia, which was due to a dural vein thrombosis (Zeki, 1991). Due to the extensive research on this patient, it is on a separate page. See page entitled "Patient LM" for more detail.
Case report #2
Another case of Akinetopsia was reported in a patient with a severe dog phobia and fluctuating walking problems and headaches. Upon further investigation, it was discovered that the patient had difficulties in perception of speed, direction, and position of objects in motion.
In October of 2003, a sixty year old male reported his inability to perceive motion. This man was struck on the head by a large, cedar light pole. He was comatose for several days, his skull was fractured and he suffered hematoma. While hunting, he was unable to see his dog move toward him or the game he was hunting would disappear before he could even aim. He was unable to see the motion to connect the appearance disappearance of animals. He eventually gave up hunting as it became dangerous - he was unable to keep track of fellow hunters. He had difficulty following his wife through a crowd. While driving, cars in front of him would appear stationary. He couldn't follow group conversations as he would lose track of who was talking. He would constantly lose his place scanning a written document in vertical and horizontal directions. He was unable to visualize 3D images depicted in 2D representations (such as a blueprint). His quality of life was quickly diminished (Pelak & Hoyt, 2005).
Case REport #3
The second case involved a 70-year-old man see a doctor with complaints of memory loss and blurred vision. Through clinical testing, no cause was identified. The patient was then evaluated for dementia. The patient was diagnosed with Alzheimer’s disease and referred for evaluation because of his complaints of visual problems. The loss of motion vision was discovered in the patient. He was forced to stop driving two years prior, as he could no longer see movement while he was driving. He reported that as an object would start to move, it would disappear completely. He was unable to judge how fast he should drive and felt that cars often “came out of nowhere.” He needed help crossing the street because he couldn’t see the cars coming. He was no longer able to watch television, especially the sports he previously enjoyed. He was still able to enjoy the news for the most part, as no significant action occurred. He also had issues finding objects located in front of him. His condition worsened exponentially as time went on. The patient developed a mild case of Simultagnosia (see page “Related Links” for more information on Simultagnosia). Optic apraxia and optic ataxia had progressed over the years of development of Alzheimer’s disease (see page “ Related Links” for more information on optic apraxia and optic ataxia). This was the first reported case of motion blindness of a patient with Alzheimer’s (Pelak & Hoyt, 2005).